Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway.

J Yuste; A Sen; L Truedsson; G Jönsson; LS Tay; C Hyams; HE Baxendale; F Goldblatt; M Botto; JS Brown

doi:10.1128/iai.00291-08

Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway.

J Yuste, A Sen, L Truedsson, G Jönsson, LS Tay, C Hyams, HE Baxendale, F Goldblatt, M Botto, JS Brown

Bristol Medical School (THS)

Research output: Contribution to journal › Article (Academic Journal) › peer-review

88 Citations (Scopus)

Original language	Undefined/Unknown
Journal	Infection and Immunity
DOIs	https://doi.org/10.1128/iai.00291-08
Publication status	Published - Aug 2008

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@article{0888c82badce491994d2c08296eb21db,

title = "Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway.",

author = "J Yuste and A Sen and L Truedsson and G J{\"o}nsson and LS Tay and C Hyams and HE Baxendale and F Goldblatt and M Botto and JS Brown",

year = "2008",

month = aug,

doi = "10.1128/iai.00291-08",

language = "Undefined/Unknown",

journal = "Infection and Immunity",

issn = "0019-9567",

publisher = "American Society for Microbiology",

}

TY - JOUR

T1 - Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway.

AU - Yuste, J

AU - Sen, A

AU - Truedsson, L

AU - Jönsson, G

AU - Tay, LS

AU - Hyams, C

AU - Baxendale, HE

AU - Goldblatt, F

AU - Botto, M

AU - Brown, JS

PY - 2008/8

Y1 - 2008/8

UR - http://europepmc.org/abstract/med/18541650

U2 - 10.1128/iai.00291-08

DO - 10.1128/iai.00291-08

M3 - Article (Academic Journal)

C2 - 18541650

SN - 0019-9567

JO - Infection and Immunity

JF - Infection and Immunity

ER -

Impaired opsonization with C3b and phagocytosis of Streptococcus pneumoniae in sera from subjects with defects in the classical complement pathway.

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