Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation

S.J Clarke; I Khaliulin; M Das; J.E Parker; K.J Heesom; A.P Halestrap

doi:10.1161/CIRCRESAHA.107.167072

Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation

S.J Clarke, I Khaliulin, M Das, J.E Parker, K.J Heesom, A.P Halestrap

Research output: Contribution to journal › Article (Academic Journal) › peer-review

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Translated title of the contribution	Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation
Original language	English
Pages (from-to)	1082 - 1090
Number of pages	9
Journal	Circulation Research
Volume	102
Issue number	9
DOIs	https://doi.org/10.1161/CIRCRESAHA.107.167072
Publication status	Published - May 2008

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10.1161/CIRCRESAHA.107.167072

http://circres.ahajournals.org/cgi/content/full/102/9/1082

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Clarke, S. J., Khaliulin, I., Das, M., Parker, J. E., Heesom, K. J., & Halestrap, A. P. (2008). Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation. Circulation Research, 102 (9), 1082 - 1090. https://doi.org/10.1161/CIRCRESAHA.107.167072

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title = "Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation",

author = "S.J Clarke and I Khaliulin and M Das and J.E Parker and K.J Heesom and A.P Halestrap",

year = "2008",

month = may,

doi = "10.1161/CIRCRESAHA.107.167072",

language = "English",

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pages = "1082 -- 1090",

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Clarke, SJ, Khaliulin, I, Das, M, Parker, JE, Heesom, KJ & Halestrap, AP 2008, 'Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation', Circulation Research, vol. 102 , no. 9, pp. 1082 - 1090. https://doi.org/10.1161/CIRCRESAHA.107.167072

Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation. / Clarke, S.J; Khaliulin, I; Das, M et al.

In: Circulation Research, Vol. 102 , No. 9, 05.2008, p. 1082 - 1090.

Research output: Contribution to journal › Article (Academic Journal) › peer-review

TY - JOUR

T1 - Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation

AU - Clarke, S.J

AU - Khaliulin, I

AU - Das, M

AU - Parker, J.E

AU - Heesom, K.J

AU - Halestrap, A.P

PY - 2008/5

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U2 - 10.1161/CIRCRESAHA.107.167072

DO - 10.1161/CIRCRESAHA.107.167072

M3 - Article (Academic Journal)

C2 - 18356542

VL - 102

SP - 1082

EP - 1090

JO - Circulation Research

JF - Circulation Research

SN - 0009-7330

IS - 9

ER -

Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation

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