Meningococcal disease associated with an acute post-streptococcal complement deficiency

Nikolaos Daskas, Katie Farmer, Richard Coward, Michel Erlewyn-Lajeunesse

Research output: Contribution to journalArticle (Academic Journal)peer-review

5 Citations (Scopus)


Chronic deficiencies in the complement pathway proteins are associated with an increased risk of meningococcal disease. Such deficiencies are caused by primary congenital immunodeficiency of a complement protein, properdin or mannose binding lectin, or are secondary to consumption of complement by systemic lupus erythematosus (SLE) or membranoproliferative glomerulonephritis (MPGN). Whatever the cause, the complement deficiency is always chronic. Here we report a case of meningococcal disease (MCD) in a child with a transient complement deficiency (CD), caused by post-streptococcal glomerulonephritis (PSGN).
Original languageEnglish
Pages (from-to)747-9
Number of pages3
JournalPediatric Nephrology
Issue number5
Publication statusPublished - 2007


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  • MRC Award

    Coward , R. J. M.


    Project: Research

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